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The commenter indicated that this conclusion was based on the limited weight gain or lack of weight gain found in animals given these steroids compared to control animals not exposed to the steroidsor the results of the two-week in-between study. This interpretation was supported by the large increase in the weight gain in the mice treated by the steroid-free hormone regimen and by the lack of weight change in rats not exposed to the steroids. Although the two-week in-between study could have influenced the results, the fact that an overall gain in weight of approximately 2.5–3% was observed in the steroid-treated rats suggests that the study was not performed adequately (see Discussion). Further, the reduction in the weight gain was observed when both groups of mice were injected with the steroid but with different doses. In contrast, no change in weight was observed in the steroid-treated rats when both groups of animals were given a single dose of 0.4 µg/kg i.p. The lack of weight gain in the two-group control animals was also not unexpected in view of the fact that the rats were treated by oral gavage. While we cannot preclude the possibility that some differences could have arisen due to the differences in the doses administered, it is evident that the differences in the results observed in one experiment may have reflected a range of possible doses and time periods and were most likely due to differences in the experimental conditions used and animals used. The only significant differences in weights observed between the groups were noted in the rats and mice treated by the steroid-free drug regimen, whereas no significant weight gain was observed in rats not treated by this regimen. This is not surprising, as rats and mice are more similar with respect to their metabolic profiles and immune system responses than other species which may be responsible for most of the differences in the results reported . It should be noted, however, that the effects of steroids on appetite and weight gain may differ according to different animals, such as mice and rats, which are more similar to each other than other species, such as humans and pigs . Therefore a greater weight gain may be observed in animals that are more similar to one another in terms of their metabolic profiles and immune system responses than in humans and other species because of these differences in responses to these treatments. In humans and other animals, the difference in responses to steroids may be due to differences in the magnitude of the effects . The differences in the effects of the steroids on appetite and weight gain may, therefore, partly reflect differences in what species are exposed to them. In contrast, these studies are not designed to evaluate the effects of different doses of steroids on feeding behavior, and the animals were given Related Article: